Why We Get Shorter As We Age: How Manual Therapy and Yoga Stretches Can Help You Stand Tall
Article At A Glance
Discs dehydrate and lose elasticity as we age, and the spinal column’s length can shrink by three centimeters or more. Thus, most of us get shorter as we age. To resist gravity’s compressive forces, the myoskeletal method uses graded exposure stretches to ease protective guarding, neurologically awaken anti-gravity muscles, and help restore lost body height. In addition, corrective exercises, including yoga, also aid in reducing protective muscle guarding and restoring body height as water and nutrients are pumped into injured and spasmodic muscles, ligaments, and intervertebral discs. Not only will your low back benefit, but so will your golf swing and your ability to walk, run, climb, lift your children, and stand tall.
The intervertebral discs are responsible for 25 percent of our spine’s overall length. Since the average adult spine measures 24 to 28 inches, the intervertebral discs account for six to seven inches. The water-filled, gel-like nucleus pulposus and annulus fibrosis are firm and flexible in young individuals. However, the vertebral discs often begin showing degenerative changes by age 40—earlier than any other connective tissue (Image 1). (1)
Image 1: Disc Degeneration Beginning at Age 40
Aging and Disc Degeneration
As discs dehydrate and lose elasticity, the spinal column’s length can shrink by three centimeters or more. Thus, most of us get shorter as we age. To resist gravity’s compressive forces, the myoskeletal method uses graded exposure stretches to ease protective guarding, neurologically awaken anti-gravity muscles, and help restore lost body height (Image 2).
Image 2 (left): A tent-like tensegrity arrangement acts as the spine’s primary shock absorber.
In a study titled “Nutrition of the Intervertebral Disc”, a team of researchers from Oxford University found that loss of nutrient supply can lead to cell death, as well as an increase in matrix degradation and disc degeneration. (2)
Vertebral discs are among the few human tissues that do not possess an independent blood supply. Even our teeth, nails, and hair have designated nutritional vessels, but the discs do not. The annular fibers require glucose for survival, and the nucleus obtains fluids and eliminates waste—primarily lactic acid—through a complicated process called imbibement. During normal daily activities, water imbibing occurs as individual spinal segments flex, extend, bend, and rotate.
Contrary to popular thinking, vertebral discs are not designed to act as the spine’s primary shock absorber. This role belongs to a marvelously designed musculofascial spring system. Notice in Image 2 how the flexible colored bands, which represent muscles and ligaments, form a tent-like tensegrity arrangement. When healthy, these soft tissues can maintain the separation of vertebral bodies even with the disc completely removed. Equipped with these facts, we’re left with this question: If restoring anti-gravity function is the key to slowing spinal disc aging, what’s the best way to pump those discs up?
Fetal Positioning and Imbibement
When picturing an intervertebral disc, most think of the outer annular fibers as circular connecting rings, similar to a radial tire, which support the gel-filled (hubcap) nucleus. However, many of the disc’s concentric tree-ring fibers do not connect, nor is the disc round (Image 3).
Image 3: The annulus resembles a radial tire encapsulating the (hubcap) nucleus.
Mother Nature cleverly thickened the anterior and lateral sides, leaving the thinner posterior fibers to absorb lubricating fluids during daily activities and also during sleep. Eighty percent of this fluid imbibement occurs during the first hour of sleep. That’s why researchers believe the best resting position for rehydrating discs is a side-lying fetal position with knees flexed, chin tucked, and lumbar lordosis flattened. This trunk-and hip-flexed fetal position opens the posterior compartment, allowing the thinner disc endplates to suck in water to nourish the disc.
Image 4: To aid in disc imbibement, the client pulls the knees to the chest and tucks the chin. The therapist’s left hand gently rocks the client so his right hand can come under the sacrum. The client is asked to perform slow pelvic tilts to hydrate discs.
The downside to this type of disc design is the increased risk of disc herniation due to a lack of posterior fiber integrity. (3) Still, there’s hope for damaged discs. A 2017 study found that conservative care fosters the drawing in of some disc bulges and may help relieve sciatic nerve root compression. (3) Gentle pain-free hyperextension exercises, such as Bhujangasana (Cobra Pose), are often used to relieve posterior nerve root compression by moving the nucleus anteriorly. However, I have experienced greater success performing fetal tractioning maneuvers and client-assisted stretches, such as the ones demonstrated in Images 4-6. Delivered throughout a series of sessions, these lumbar-pumping maneuvers appear to increase fluid absorption and aid in body height restoration.
Image 5: The client grasps the top of the therapy table. The therapist’s left-hand snakes behind his back to secure client’s right hip while his right forearm hooks the lateral fascia. The client is asked to pull down on the therapy table against the therapist’s resistance to a count of five and then relax. To decompress discs and stimulate anti-gravity muscles, the therapist’s forearm lifts the thorax while his left-hand resists. Repeat.
Recently, I’ve begun using an accurate measuring device to track client height during intake and again after several sessions. I find this tracking routine acts as a novel stimulus that helps keep both client and therapist engaged in the process. To enhance the therapeutic outcome, I encourage clients to perform a variety of movements on their own time, from swimming and mini-trampoline bouncing to tai chi and hiking. These playful activities, when combined with myoskeletal therapy, bring awareness to movement “blind spots” and help clients break non-optimal movement patterns.
Image 6: The client grasps the top of the therapy table. The therapist’s soft palms meet on the lateral ribcage and hook the fascia. The client pulls down on the therapy table against the therapist’s resistance to a count of five and then relaxes. The therapist lifts the thorax to decompress the vertebral discs. Repeat.
How Yoga and Manual Therapy Keep Our Discs Hydrated
In sports and recreation, a healthy anti-gravity musculofascial system transmits and releases stored elastic energy from the ground up through the individual spinal segments, causing them to spring open and hydrate the vertebral discs. To relieve protective muscle guarding, restore anti-gravity function, and bring some lift to the body, I’ve found success using therapeutic stretching and manual spinal tractioning in a controlled, comfortable manner.
Corrective exercise also aids in reducing protective muscle guarding and restoring body height as water and nutrients are pumped into injured and spasmodic muscles, ligaments, and intervertebral discs. Not only will your low back benefit, but so will your golf swing and your ability to walk, run, climb, lift your children, and stand tall.
Reprinted with permission from Erik Dalton. Images courtesy of Erik Dalton.
Erik Dalton, Ph.D., is executive director of the Freedom From Pain Institute, creator of Myoskeletal Alignment Techniques, and author of three best-selling manual therapy textbooks and online home-study programs. Educated in massage, osteopathy, and Rolfing, he resides in Oklahoma City, Oklahoma and San Jose, Costa Rica. View his articles and videos at www.erikdalton.com or Facebook’s Erik Dalton Techniques Group.
Resources
1. Urban, J., & Roberts, S. (2003). Degeneration of the intervertebral disc. Arthritis Research & Therapy, 5(3), 120-130.
2. Urban J.P., Smith, S., & Fairbank, J.C. (2004). Nutrition of the intervertebral disc. Spine, 29( 23), 2700-2709.
3. Altun, I., & Yüksel, K.Z. (2017). Lumbar herniated disc: spontaneous regression. Korean Journal of Pain, 30 (1), 44-50.